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Schwemmle

Prof. Dr.
Martin Schwemmle

Institute of Virology
Phone: 0049-761-203-6526

www.uniklinik-freiburg.de/virologie-en/research/research-teams

CV

  • 1992-1994 Postdoctoral Fellow at the Institute of Virology, Medical Center – University of Freiburg
  • 1995-1997 Postdoctoral Fellow at the Institute of Neurovirology, UC-Irvine, USA            
  • 1997-2001 Research Associate at the Institute of Virology, Medical Center – University of Freiburg
  • 2002-2003 Tenured Research Associate at the Institute for Virology, University of Zürich, Switzerland
  • Since 2003 Professor (C3) at the Institute of Virology, Medical Center - University of Freiburg

Focus of research

  • Zoonotic virus infections
  • Innate Immunity
  • Molecular Biology of RNA viruses

Selected publications

  • Moreira ÉA, Weber A, Bolte H, Kolesnikova L, Giese S, Lakdawala S, Beer M, Zimmer G, García-Sastre A, Schwemmle M, Juozapaitis M. A conserved infl uenza A virus nucleoprotein code controls specifi c viral genome packaging. Nat Commun. 2016; 7:12861.
  • Moreira ÉA, Locher S, Kolesnikova L, Bolte H, Aydillo T, García-Sastre A, Schwemmle M, Zimmer G. Synthetically derived bat influenza A-like viruses reveal a cell type- but not species-specifi c tropism. Proc Natl Acad Sci USA. 2016; 113:12797-12802.
  • Juozapaitis M, Aguiar Moreira É, Mena I, Giese S, Riegger D, Pohlmann A, Höper D, Zimmer G, Beer M, García-Sastre A, Schwemmle M. An infectious bat-derived chimeric infl uenza virus harbouring the entry machinery of an infl uenza A virus. Nat Commun. 2014; 5:4448.
  • Wu YJ, Schulz H, Lin CC, Saar K, Patone G, Fischer H, Hübner N, Heimrich B, Schwemmle M. Borna disease virus-induced neuronal degeneration dependent on host genetic background and prevented by soluble factors. Proc Natl Acad Sci USA. 2013; 110(5):1899-904.
  • Mänz B, Brunotte L, Reuther P, Schwemmle M. Adaptive mutations in NEP compensate for defective H5N1 RNA replication in cultured human cells. Nat Commun. 2012; 3:802.

Spezielle Forschungsmethoden

  • In vitro and in vivo infection models
  • Reverse genetic of Influenza viruses
  • Research with highly pathogenic viruses (BSL3 level)