Michael Köttgen

Department Innere Medizin
Klinik für Innere Medizin IV

Tel.: 0761/270-32990
michael.koettgen@uniklinik-freiburg.de
www.nephrolab.org/groups/michael-koettgen

1997-1998 Arzt im Praktikum, Department Innere Medizin, Universitätsklinikum Freiburg
1998-2004 Postdoc, Physiologisches Institut und Department Innere Medizin, Universität Freiburg
2004-2009 Postdoc - Research Associate, Department of Biological Chemistry, Johns Hopkins University, Baltimore, USA
2010 Adjunct Professor, Department of Medicine, Johns Hopkins University, School of Medicine, Baltimore, USA
Seit 2010 W3-Professor für Nephrologie, Universitätsklinikum Freiburg

Hofherr A, Köttgen M. Polycystic kidney disease: Cilia and mechanosensation revisited. Nat Rev Nephrol 12, 2016; 318-319.
Hofherr A, Wagner CJ, Watnick T, Köttgen M. Targeted rescue of a polycystic kidney disease mutation by lysosomal inhibition. Kidney Int. 2016; 89,949-955.
Hofherr A, Wagner C, Fedeles S, Somlo S, Köttgen M. N-glycosylation determines the abundance of the transient receptor potential channel TRPP2. J Biol Chem. 2014; 289, 14854-14867.
Wegierski T, Steffl D, Kopp C, Tauber R, Buchholz B, Nitschke R, Kuehn EW, Walz G, Köttgen M. TRPP2 channels regulate apoptosis through the Ca2+ concentration in the endoplasmic reticulum. EMBO J. 2009; 28, 490-499.
Woodward OM, Köttgen A, Coresh J, Boerwinkle E, Guggino WB, Köttgen M. Identification of a urate transporter, ABCG2, with a common functional polymorphism causing gout. Proc Natl Acad Sci U S A. 2009; 106,10338-10342.